1 |
Ramseier CA, Eick S, Brönnimann C, et al. Host-derived biomarkers at teeth and implants in partially edentulous patients. A 10-year retrospective study[J]. Clin Oral Implants Res, 2016, 27(2): 211-217.
|
2 |
Lee CT, Huang YW, Zhu L, et al. Prevalences of peri-implantitis and peri-implant mucositis: systematic review and meta-analysis[J]. J Dent, 2017, 62: 1-12.
|
3 |
Schwarz F, Derks J, Monje A, et al. Peri-implantitis[J]. J Periodontol, 2018, 89(): S267-S290.
|
4 |
Schwendicke F, Tu YK, Stolpe M. Preventing and treating peri-implantitis: a cost-effectiveness analysis[J]. J Periodontol, 2015, 86(9): 1020-1029.
|
5 |
Salvi GE, Cosgarea R, Sculean A. Prevalence and mechanisms of peri-implant diseases[J]. J Dent Res, 2017, 96(1): 31-37.
|
6 |
Dabdoub SM, Tsigarida AA, Kumar PS. Patient-specific analysis of periodontal and peri-implant microbiomes[J]. J Dent Res, 2013, 92(12): 168s-175s.
|
7 |
Hallström H, Persson GR, Lindgren S, et al. Systemic antibiotics and debridement of peri-implant mucositis. A randomized clinical trial[J]. J Clin Periodontol, 2012, 39(6): 574-581.
|
8 |
Tonetti MS, Chapple IL, Jepsen S, et al. Primary and secondary prevention of periodontal and peri-implant diseases: introduction to, and objectives of the 11th European Workshop on Periodontology consensus conference[J]. J Clin Periodontol, 2015, 42(): S1-S4.
|
9 |
Heitz-mayfield LJ. Peri-implant diseases: diagnosis and risk indicators[J]. J Clin Periodontol, 2008, 35(8): 292-304.
|
10 |
Almehmadi AH, Alghamdi F. Biomarkers of alveolar bone resorption in gingival crevicular fluid: a systematic review[J]. Arch Oral Biol, 2018, 93: 12-21.
|
11 |
Sorsa T, Gieselmann D, Arweiler NB, et al. A quantitative point-of-care test for periodontal and dental peri-implant diseases[J]. Nat Rev Dis Primers, 2017, 3: 17069.
|
12 |
Serino G, Turri A, Lang NP. Probing at implants with peri-implantitis and its relation to clinical peri-implant bone loss[J]. Clin Oral Implants Res, 2013, 24(1): 91-95.
|
13 |
Renvert S, Persson GR, Pirih FQ, et al. Peri-implant health, peri-implant mucositis, and peri-implantitis: case definitions and diagnostic considerations[J]. J Periodontol, 2018, 89(): S304-S312.
|
14 |
Belibasakis GN, Bostanci N, Marsh PD, et al. Applications of the oral microbiome in personalized dentistry[J]. Arch Oral Biol, 2019, 104: 7-12.
|
15 |
Biomarkers Definitions Working Group. Biomarkers and surrogate endpoints: preferred definitions and conceptual framework[J]. Clin Pharmacol Ther, 2001, 69(3): 89-95.
|
16 |
Bhavsar I, Miller CS, Ebersole JL, et al. Biological response to peri-implantitis treatment[J]. J Periodontal Res, 2019, 54(6): 720-728.
|
17 |
Butler GS, Overall CM. Matrix metalloproteinase processing of signaling molecules to regulate inflammation[J]. Periodontol 2000, 2013, 63(1): 123-148.
|
18 |
Yuan S, Lin LS, Gan RH, et al. Elevated matrix metalloproteinase 7 expression promotes the proliferation, motility and metastasis of tongue squamous cell carcinoma[J]. BMC Cancer, 2020, 20(1): 33.
|
19 |
Leeman MF, Curran S, Murray GI. New insights into the roles of matrix metalloproteinases in colorectal cancer development and progression[J]. J Pathol, 2003, 201(4): 528-534.
|
20 |
Uitto VJ, Salonen JI, Firth JD, et al. Matrilysin (matrix metalloproteinase-7) expression in human junctional epithelium[J]. J Dent Res, 2002, 81(4): 241-246.
|
21 |
Emingil G, Tervahartiala T, Mãntylã P, et al. Gingival crevicular fluid matrix metalloproteinase (MMP)-7, extracellular MMP inducer, and tissue inhibitor of MMP-1 levels in periodontal disease[J]. J Periodontol, 2006, 77(12): 2040-2050.
|
22 |
Lundmark A, Davanian H, Båge T, et al. Transcriptome analysis reveals mucin 4 to be highly associated with periodontitis and identifies pleckstrin as a link to systemic diseases[J]. Sci Rep, 2015, 5: 18475.
|
23 |
Lundmark A, Johannsen G, Eriksson K, et al. Mucin 4 and matrix metalloproteinase 7 as novel salivary biomarkers for periodontitis[J]. J Clin Periodontol, 2017, 44(3): 247-254.
|
24 |
Papapanou PN, Sanz M, Buduneli N, et al. Periodontitis: consensus report of workgroup 2 of the 2017 World Work-shop on the Classification of Periodontal and Peri-Implant Diseases and Conditions[J]. J Periodontol, 2018, 89(): S173-S182.
|
25 |
Fu X, Kassim SY, Parks WC, et al. Hypochlorous acid oxygenates the cysteine switch domain of pro-matrilysin (MMP-7). A mechanism for matrix metalloproteinase activation and atherosclerotic plaque rupture by myeloperoxidase[J]. J Biol Chem, 2001, 276(44): 41279-41287.
|
26 |
Ho BY, Wu YM, Chang KJ, et al. Dimerumic acid inhibits SW620 cell invasion by attenuating H2O2-mediated MMP-7 expression via JNK/C-Jun and ERK/C-Fos activation in an AP-1-dependent manner[J]. Int J Biol Sci, 2011, 7(6): 869-880.
|
27 |
Kiili M, Cox SW, Chen HY, et al. Collagenase-2 (MMP-8) and collagenase-3 (MMP-13) in adult periodontitis: molecular forms and levels in gingival crevicular fluid and immunolocalisation in gingival tissue[J]. J Clin Periodontol, 2002, 29(3): 224-232.
|
28 |
de Oliveira Nóbrega FJ, de Oliveira D, Vasconcelos RG, et al. Study of the participation of MMP-7, EMMPRIN and cyclophilin A in the pathogenesis of periodontal disease[J]. Arch Oral Biol, 2016, 72: 172-178.
|
29 |
Sánchez GA, Miozza V, Delgado A, et al. Determination of salivary levels of mucin and amylase in chronic periodontitis patients[J]. J Periodontal Res, 2011, 46(2): 221-
|
227 |
|
30 |
焦鹏. MMP-7和MUC-4在慢性牙周炎中的表达及临床意义[D]. 长春: 吉林大学, 2018.
|
|
Jiao P. Expression and clinical significance of MMP-7 and MUC-4 in chronic periodontitis[D]. Changchun: Jilin University, 2018.
|
31 |
Hanemaaijer R, Sorsa T, Konttinen YT, et al. Matrix metalloproteinase-8 is expressed in rheumatoid synovial fibroblasts and endothelial cells. Regulation by tumor necrosis factor-alpha and doxycycline[J]. J Biol Chem, 1997, 272(50): 31504-31509.
|
32 |
Song HK, Noh EM, Kim JM, et al. Reversine inhibits MMP-3, IL-6 and IL-8 expression through suppression of ROS and JNK/AP-1 activation in interleukin-1β-stimulated human gingival fibroblasts[J]. Arch Oral Biol, 2019, 108: 104530.
|
33 |
Alfakry H, Malle E, Koyani CN, et al. Neutrophil proteolytic activation cascades: a possible mechanistic link between chronic periodontitis and coronary heart disease[J]. Innate Immun, 2016, 22(1): 85-99.
|